Pentoxifylline

ATC Code C04AD03

Pentoxifylline

Definition

A methylxanthine derivative with hemorrheologic and immunomodulating properties. Pentoxifylline inhibits phosphodiesterase, resulting in increased levels of cyclic adenosine monophosphate (cAMP) in erythrocytes, endothelium, and the surrounding tissues. This leads to vasodilation, improves erythrocyte flexibility, and enhances blood flow. In addition, the increased level of cAMP in platelets inhibits platelet aggregation, which may contribute to a reduction in blood viscosity. This agent also inhibits production of tumor necrosis factor-alpha and interferon-gamma, while it induces Th2-like (T-helper 2) cytokine production, thereby inhibiting Th1-mediated (T-helper 1) inflammatory and autoimmune responses.

Also known as 3,7-Dihydro-3,7-dimethyl-1-(5-oxohexyl)-1H-purine-2,6-dione, Oxpentifylline, PENTOXIFYLLINE, Pentoxyphylline, PTX, Trental — per NCIT

Also identified as

Indications

Disease Relation Source
Brain ischemia may treat MEDRT · Public domain (U.S. Government work)
Cerebral malaria may treat MEDRT · Public domain (U.S. Government work)
Diabetic neuropathy may treat MEDRT · Public domain (U.S. Government work)
Diabetic peripheral angiopathy may treat MEDRT · Public domain (U.S. Government work)
Intermittent vascular claudication may treat MEDRT · Public domain (U.S. Government work)
Stroke may treat MEDRT · Public domain (U.S. Government work)
Thrombotic disease may treat MEDRT · Public domain (U.S. Government work)
Vasculitis may treat MEDRT · Public domain (U.S. Government work)
Vasculogenic impotence may treat MEDRT · Public domain (U.S. Government work)

Organ Effects

Organ Side effect / interaction Source
Brain may treat a disease of this organ DERIVED · CC BY 4.0
Cardiovascular system may treat a disease of this organ DERIVED · CC BY 4.0
Central nervous system may treat a disease of this organ DERIVED · CC BY 4.0
Nervous system may treat a disease of this organ DERIVED · CC BY 4.0

4 entries above are marked DERIVED: Atlas Médico inferred them by combining two sourced claims, and no source asserts these links directly. Treat them as a navigation aid, not as a clinical statement.